Growing numbers of inserted dental implants correlate to an increasing number of infected implants1. Periimplantitis (PI) is defined as an inflammatory process affecting both periimplant soft and hard tissue. Craterlike bone defects, ongoing bone loss, pus, and bleeding on probing (BoP) are clinical parameters that have to be present to justify the diagnosis of PI 2,3. PI correlates to bacterial biofilms growing on implants or abutments4. In view of the various definitions of PI and the lack of
agreement in the dental community about an acceptable threshold of bone loss, there is no consensus on when pathology starts and how PI can be diagnosed precisely. Hence, there is no consensus about prevalence data5,6,7. A proper treatment modality is lacking8. Whether the bacterial biofilm is the single causal factor or only correlates to PI9 is still a matter of discussion. This debate about etiology is not merely an academic question, but may influence the success rate of therapy, because of possible differences in specimen susceptibility and uncorrectable surgical or mechanical obstacles. In any event, biofilm needs to be removed to prevent progression of disease, or to treat PI successfully.
J. Clin. Med. 2019, 8, 1397; doi:10.3390/jcm8091397